 Kevin Magnaye, PhD
 Biography | University of Chicago, Chicago, IL | PhD | 12/2020 | Human Genetics | | University of Chicago, Chicago, IL | MS | 09/2017 | Human Genetics | | University of Washington, Seattle, WA | BA | 06/2015 | Anthropology | | University of Washington, Seattle, WA | BS | 06/2015 | Biology |
Overview Kevin received undergraduate degrees in Biology and Anthropology at the University of Washington Seattle and a PhD in Human Genetics at the University of Chicago. His doctoral thesis focused on identifying and characterizing genetic and epigenetic mechanisms underlying maternal transmission of asthma risk. He served as a Ruth L. Kirschstein Predoctoral Fellow and received a Graduate Global Impact Scholarship to lead a research study on the genetics of infection at the Pasteur Institute in Paris, France. As a postdoctoral scholar in the Lynch lab at UCSF, he is studying host-microbe interactions in early life and their impact on health and disease throughout the lifespan. His broad scientific goal is to understand the etiology of biological phenomena and transform basic science findings into meaningful diagnostic and therapeutic solutions. He is also passionate about outreach and mentorship and has served as a coordinator for the UW Genomics for Minorities Project and as an instructor for high school and undergraduate students in Phoenix, Arizona and in Oaxaca, Mexico as part of the Clubes de Ciencia initiative.
Research ORNG Applications Bibliographic
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A-to-I editing of miR-200b-3p in airway cells is associated with moderate-to-severe asthma. Eur Respir J. 2021 Jan 14.
Magnaye KM, Naughton KA, Huffman J, Hogarth DK, Naureckas ET, White SR, Ober C. PMID: 33446603.
View in: PubMed Mentions: Fields:
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Sex-specific differences in peripheral blood leukocyte transcriptional response to LPS are enriched for HLA region and X chromosome genes. Sci Rep. 2021 Jan 13; 11(1):1107.
Stein MM, Conery M, Magnaye KM, Clay SM, Billstrand C, Nicolae R, Naughton K, Ober C, Thompson EE. PMID: 33441806.
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Expression quantitative trait locus fine mapping of the 17q12-21 asthma locus in African American children: a genetic association and gene expression study. Lancet Respir Med. 2020 05; 8(5):482-492.
Ober C, McKennan CG, Magnaye KM, Altman MC, Washington C, Stanhope C, Naughton KA, Rosasco MG, Bacharier LB, Billheimer D, Gold DR, Gress L, Hartert T, Havstad S, Khurana Hershey GK, Hallmark B, Hogarth DK, Jackson DJ, Johnson CC, Kattan M, Lemanske RF, Lynch SV, Mendonca EA, Miller RL, Naureckas ET, O'Connor GT, Seroogy CM, Wegienka G, White SR, Wood RA, Wright AL, Zoratti EM, Martinez FD, Ownby D, Nicolae DL, Levin AM, Gern JE, Environmental Influences on Child Health Outcomes-Children's Respiratory Research Workgroup . PMID: 32380068.
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Parent of origin gene expression in a founder population identifies two new candidate imprinted genes at known imprinted regions. PLoS One. 2018; 13(9):e0203906.
Mozaffari SV, Stein MM, Magnaye KM, Nicolae DL, Ober C. PMID: 30204804.
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Complex signatures of natural selection at GYPA. Hum Genet. 2018 Feb; 137(2):151-160.
Bigham AW, Magnaye K, Dunn DM, Weiss RB, Bamshad M. PMID: 29362874.
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A decade of research on the 17q12-21 asthma locus: Piecing together the puzzle. J Allergy Clin Immunol. 2018 09; 142(3):749-764.e3.
Stein MM, Thompson EE, Schoettler N, Helling BA, Magnaye KM, Stanhope C, Igartua C, Morin A, Washington C, Nicolae D, Bønnelykke K, Ober C. PMID: 29307657.
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Estimates of continental ancestry vary widely among individuals with the same mtDNA haplogroup. Am J Hum Genet. 2015 Feb 05; 96(2):183-93.
Emery LS, Magnaye KM, Bigham AW, Akey JM, Bamshad MJ. PMID: 25620206.
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Multiple mutant T alleles cause haploinsufficiency of Brachyury and short tails in Manx cats. Mamm Genome. 2013 Oct; 24(9-10):400-8.
Buckingham KJ, McMillin MJ, Brassil MM, Shively KM, Magnaye KM, Cortes A, Weinmann AS, Lyons LA, Bamshad MJ. PMID: 23949773.
View in: PubMed Mentions: 6 Fields: Translation: AnimalsCells
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| Year | Publications |
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| 2013 | 1 | | 2015 | 1 | | 2018 | 3 | | 2020 | 1 | | 2021 | 2 |
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This graph shows the number and percent of publications by field.
Fields are based on how the National Library of Medicine (NLM) classifies the publications' journals and might not represent the specific topics of the publications.
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