 Tanvi Sinha, PhD
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| Title(s) | Associate Specialist, Cardiovascular Research Inst |
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| School | School of Medicine |
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| Address | 555 Mission Bay Blvd South
San Francisco CA 94158
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| Phone | 415-502-7591 |
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ORCID
.gif) | 0000-0001-9509-5687  |
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| vCard | Download vCard |
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 Bibliographic
Altmetrics Details
PMC Citations indicate the number of times the publication was cited by articles in PubMed Central, and the Altmetric score represents citations in news articles and social media.
(Note that publications are often cited in additional ways that are not shown here.)
Fields are based on how the National Library of Medicine (NLM) classifies the publication's journal and might not represent the specific topic of the publication.
Translation tags are based on the publication type and the MeSH terms NLM assigns to the publication.
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Cardiovascular development and survival require Mef2c function in the myocardial but not the endothelial lineage. Dev Biol. 2019 01 15; 445(2):170-177.
Materna SC, Sinha T, Barnes RM, Lammerts van Bueren K, Black BL. PMID: 30521808.
View in: PubMed Mentions: Fields: Translation: Animals
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Cooperative activation of cardiac transcription through myocardin bridging of paired MEF2 sites. Development. 2017 04 01; 144(7):1235-1241.
Anderson CM, Hu J, Thomas R, Gainous TB, Celona B, Sinha T, Dickel DE, Heidt AB, Xu SM, Bruneau BG, Pollard KS, Pennacchio LA, Black BL. PMID: 28351867.
View in: PubMed Mentions: 3 Fields: Translation: AnimalsCells
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Spatial regulation of cell cohesion by Wnt5a during second heart field progenitor deployment. Dev Biol. 2016 Apr 01; 412(1):18-31.
Li D, Sinha T, Ajima R, Seo HS, Yamaguchi TP, Wang J. PMID: 26916252.
View in: PubMed Mentions: 4 Fields: Translation: AnimalsCells
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MEF2C regulates outflow tract alignment and transcriptional control of Tdgf1. Development. 2016 Mar 01; 143(5):774-9.
Barnes RM, Harris IS, Jaehnig EJ, Sauls K, Sinha T, Rojas A, Schachterle W, McCulley DJ, Norris RA, Black BL. PMID: 26811383.
View in: PubMed Mentions: 11 Fields: Translation: HumansAnimalsCells
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Loss of tumor suppressive microRNA-31 enhances TRADD/NF-?B signaling in glioblastoma. Oncotarget. 2015 Jul 10; 6(19):17805-16.
Rajbhandari R, McFarland BC, Patel A, Gerigk M, Gray GK, Fehling SC, Bredel M, Berbari NF, Kim H, Marks MP, Meares GP, Sinha T, Chuang J, Benveniste EN, Nozell SE. PMID: 26164206.
View in: PubMed Mentions: 14 Fields: Translation: HumansAnimalsCells
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Mapping the dynamic expression of Wnt11 and the lineage contribution of Wnt11-expressing cells during early mouse development. Dev Biol. 2015 Feb 15; 398(2):177-92.
Sinha T, Lin L, Li D, Davis J, Evans S, Wynshaw-Boris A, Wang J. PMID: 25448697.
View in: PubMed Mentions: 6 Fields: Translation: AnimalsCells
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Loss of Wnt5a disrupts second heart field cell deployment and may contribute to OFT malformations in DiGeorge syndrome. Hum Mol Genet. 2015 Mar 15; 24(6):1704-16.
Sinha T, Li D, Théveniau-Ruissy M, Hutson MR, Kelly RG, Wang J. PMID: 25410658.
View in: PubMed Mentions: 13 Fields: Translation: AnimalsCells
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Disheveled mediated planar cell polarity signaling is required in the second heart field lineage for outflow tract morphogenesis. Dev Biol. 2012 Oct 01; 370(1):135-44.
Sinha T, Wang B, Evans S, Wynshaw-Boris A, Wang J. PMID: 22841628.
View in: PubMed Mentions: 21 Fields: Translation: AnimalsCells
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Wnt signaling in mammalian development: lessons from mouse genetics. Cold Spring Harb Perspect Biol. 2012 May 01; 4(5).
Wang J, Sinha T, Wynshaw-Boris A. PMID: 22550229.
View in: PubMed Mentions: 32 Fields: Translation: AnimalsCells
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Disruption of PCP signaling causes limb morphogenesis and skeletal defects and may underlie Robinow syndrome and brachydactyly type B. Hum Mol Genet. 2011 Jan 15; 20(2):271-85.
Wang B, Sinha T, Jiao K, Serra R, Wang J. PMID: 20962035.
View in: PubMed Mentions: 46 Fields: Translation: AnimalsCells
This graph shows the total number of publications by year. To see the data as text, click here.
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| Year | Publications |
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| 2010 | 1 | | 2012 | 2 | | 2014 | 2 | | 2015 | 1 | | 2016 | 2 | | 2017 | 1 | | 2018 | 1 |
This graph shows the number and percent of publications by field.
Fields are based on how the National Library of Medicine (NLM) classifies the publications' journals and might not represent the specific topics of the publications.
Note that an individual publication can be assigned to more than one field. As a result, the publication counts in this graph might add up to more than the number of publications the person has written.
To see the data as text, click here.
This graph shows the number and percent of publications by field.
Fields are based on how the National Library of Medicine (NLM) classifies the publications' journals and might not represent the specific topics of the publications.
Note that an individual publication can be assigned to more than one field. As a result, the publication counts in this graph might add up to more than the number of publications the person has written.
To see the data as text, click here.
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