Gonzalo Barrera-Hernandez, PhD

Address600 16th St
San Francisco CA 94158
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    I joined UCSF in 2012, following 12 ½ years at the office of Innovation, Alliances & Services (IAS) (formerly known as the Office of Technology Transfer) at the UC Office of the President, where I was responsible for all IP management and technology licensing matters of a life-sciences portfolio of technologies from several UC campuses. While at IAS I was also responsible for the first UC System Proof-of-Concept Fund, the UC System-Wide Technology Transfer Forum Program, the Technology Transfer Annual Report for the UC System, and for managing for the UC System the approval process for the acceptance of equity in licensing transactions. Prior to joining UC, I worked in technology transfer for 2 years, on detail, at the NIH.

    I received my Ph.D. in Biochemistry and Molecular Biology from the University of Calgary, Canada, where my research focused on gene regulation in Diabetes mellitus. My postdoctoral research at the NCI-NIH focused on the role of the thyroid hormone receptor in cell cycle regulation and on the mechanism controlling the switch from cell cycle regulation to differentiation. I received a degree in Biological Sciences from the University of Buenos Aires, Argentina.

    I am passionate about helping UCSF achieve its mission of advancing health worldwide through partnerships with industry, and by working closely with faculty to commercialize UCSF inventions.

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    Publications listed below are automatically derived from MEDLINE/PubMed and other sources, which might result in incorrect or missing publications. Researchers can login to make corrections and additions, or contact us for help.
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    1. Barrera-Hernandez G, Cultraro CM, Pianetti S, Segal S. Mad1 function is regulated through elements within the carboxy terminus. Mol Cell Biol. 2000 Jun; 20(12):4253-64. PMID: 10825189; PMCID: PMC85793.
    2. Barrera-Hernandez G, Park KS, Dace A, Zhan Q, Cheng SY. Thyroid hormone-induced cell proliferation in GC cells is mediated by changes in G1 cyclin/cyclin-dependent kinase levels and activity. Endocrinology. 1999 Nov; 140(11):5267-74. PMID: 10537157.
      View in: PubMed
    3. Barrera-Hernandez G, Zhan Q, Wong R, Cheng SY. Thyroid hormone receptor is a negative regulator in p53-mediated signaling pathways. DNA Cell Biol. 1998 Sep; 17(9):743-50. PMID: 9778033.
      View in: PubMed
    4. Yu CL, Driggers P, Barrera-Hernandez G, Nunez SB, Segars JH, Cheng S. The tumor suppressor p53 is a negative regulator of estrogen receptor signaling pathways. Biochem Biophys Res Commun. 1997 Oct 20; 239(2):617-20. PMID: 9344880.
      View in: PubMed
    5. Barrera-Hernandez G, Wanke IE, Wong NC. Phlorizin or vanadate treatment reverses impaired expression of albumin and hepatocyte nuclear factor 1 in diabetic rats. Diabetes. 1996 Sep; 45(9):1217-22. PMID: 8772725.
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    6. Barrera-Hernandez G, Wanke IE, Wong NC. Effects of diabetes mellitus on hepatocyte nuclear factor 1 decrease albumin gene transcription. J Biol Chem. 1996 Apr 26; 271(17):9969-75. PMID: 8626635.
      View in: PubMed
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