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Yuta Senzai, PhD

Title(s)Postdoctoral Scholar, Physiology
SchoolSchool of Medicine
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    Publications listed below are automatically derived from MEDLINE/PubMed and other sources, which might result in incorrect or missing publications. Researchers can login to make corrections and additions, or contact us for help.
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    1. Senzai Y, Fernandez-Ruiz A, Buzsáki G. Layer-Specific Physiological Features and Interlaminar Interactions in the Primary Visual Cortex of the Mouse. Neuron. 2019 02 06; 101(3):500-513.e5. PMID: 30635232.
      View in: PubMed
    2. Senzai Y. Function of local circuits in the hippocampal dentate gyrus-CA3 system. Neurosci Res. 2019 Mar; 140:43-52. PMID: 30408501.
      View in: PubMed
    3. Senzai Y, Buzsáki G. Physiological Properties and Behavioral Correlates of Hippocampal Granule Cells and Mossy Cells. Neuron. 2017 Feb 08; 93(3):691-704.e5. PMID: 28132824.
      View in: PubMed
    4. Stark E, Roux L, Eichler R, Senzai Y, Royer S, Buzsáki G. Pyramidal cell-interneuron interactions underlie hippocampal ripple oscillations. Neuron. 2014 Jul 16; 83(2):467-480. PMID: 25033186.
      View in: PubMed
    5. Tanaka K, Furuyashiki T, Kitaoka S, Senzai Y, Imoto Y, Segi-Nishida E, Deguchi Y, Breyer RM, Breyer MD, Narumiya S. Prostaglandin E2-mediated attenuation of mesocortical dopaminergic pathway is critical for susceptibility to repeated social defeat stress in mice. J Neurosci. 2012 Mar 21; 32(12):4319-29. PMID: 22442093.
      View in: PubMed
    6. Kohei Tanaka, Shiho Kitaoka, Yuta Senzai, Tomoyuki Furuyashiki, Shuh Narumiya. Prostaglandin E receptor EP1 is critical for social withdrawal and the plasticity of dopaminergic neurons after repeated social defeat. Neuroscience Research. 2011 Sep 1; 71:e167.
      View in: Publisher Site
    7. Tomoyuki Furuyashiki, Kohei Tanaka, Shiho Kitaoka, Yuta Senzai, Shuh Narumiya. Prostaglandin E2-mediated desensitization of mesocortical dopamine neurons is critical for susceptibility to repeated social defeat stress. Neuroscience Research. 2011 Sep 1; 71:e52.
      View in: Publisher Site