Regis Kelly, PhD

Title(s)Professor Emeritus, Biochemistry and Biophysics
SchoolSchool of Medicine
Address600 16th Street, #S222
San Francisco CA 94158
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    Other Positions
    Title(s)Director, QB3 Institute

    Collapse Overview 
    Collapse Overview
    Dr. Regis B. Kelly is the Director of one of four California Institutes for Science and Innovation, created by the California Legislature to strengthen the academic foundation of its technology-based industries. QB3 is the only one of the four devoted exclusively to biology and to the life science industries. It is an innovation center made up of over 200 quantitative biologists at three northern California campuses (UCB, UCSC & UCSF) working at the interface of the physical and biological sciences and a team of professionals converting its discoveries into practical benefits for society.

    From 2000 to 2004, Dr. Kelly served as Executive Vice Chancellor at the University of California in San Francisco, where his major responsibility was the new Mission Bay campus.

    From 1995 to 2000, Dr. Kelly served as Chair of the Department of Biochemistry and Biophysics at UCSF; from 1988 to 1995, he was the Director of UCSF’s Cell Biology Graduate Program; and from 1992 to 2000, he was the Director of the Hormone Research Institute at UCSF. He has published extensively in the areas of cell and neurobiology.

    Dr. Kelly received his undergraduate degree in Physics from the University of Edinburgh in Scotland in 1961 and his Ph.D. in Biophysics from the California Institute of Technology in 1967. Following a post-doctoral fellowship at Stanford, Dr. Kelly was an instructor in the Department of Neurobiology at Harvard. He has served as Chairman of the Bay Area Scientific Innovation Consortium (BASIC) and on the Boards of the Malaysian Biotechnology Industry Advisory Board, the Scleroderma Research Foundation, Bridge Pharmaceuticals, the San Francisco Mayor’s Biotechnology Advisory Group and the San Francisco China Desk, among others. He is also a General Partner of Mission Bay Capital venture fund.

    Collapse Research 
    Collapse Research Activities and Funding
    Human Subjects Research Enhancements Program (HSREP)
    NIH S07RR018260Sep 1, 2002 - Aug 31, 2005
    Role: Co-Principal Investigator
    Presynaptic Mechanisms of Neural Plasticity
    NIH P01DA010154Sep 30, 1995 - Apr 30, 2018
    Role: Co-Investigator
    NIH R13AG012952Aug 20, 1994 - Jul 31, 1995
    Role: Principal Investigator
    NIH T32GM008120Jul 1, 1985 - Jun 30, 2001
    Role: Principal Investigator
    NIH R01DK033937Apr 1, 1984 - Mar 29, 1997
    Role: Principal Investigator
    NIH R01AM033937Apr 1, 1984 - Mar 31, 1987
    Role: Principal Investigator
    Function and development in the synapse
    NIH P01NS016033Apr 1, 1980 - Jun 30, 2009
    Role: Co-Investigator
    Antibodies to Synaptic Vesicles
    NIH R01NS015927Dec 1, 1979 - Jun 30, 2006
    Role: Principal Investigator
    NIH R01NS009878Sep 1, 1978 - Apr 30, 2004
    Role: Principal Investigator

    Collapse Bibliographic 
    Collapse Publications
    Publications listed below are automatically derived from MEDLINE/PubMed and other sources, which might result in incorrect or missing publications. Researchers can login to make corrections and additions, or contact us for help. to make corrections and additions.
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    Altmetrics Details PMC Citations indicate the number of times the publication was cited by articles in PubMed Central, and the Altmetric score represents citations in news articles and social media. (Note that publications are often cited in additional ways that are not shown here.) Fields are based on how the National Library of Medicine (NLM) classifies the publication's journal and might not represent the specific topic of the publication. Translation tags are based on the publication type and the MeSH terms NLM assigns to the publication. Some publications (especially newer ones and publications not in PubMed) might not yet be assigned Field or Translation tags.) Click a Field or Translation tag to filter the publications.
    1. Dap160/intersectin scaffolds the periactive zone to achieve high-fidelity endocytosis and normal synaptic growth. Neuron. 2004 Jul 22; 43(2):207-19. Marie B, Sweeney ST, Poskanzer KE, Roos J, Kelly RB, Davis GW. PMID: 15260957.
      View in: PubMed   Mentions: 123     Fields:    Translation:AnimalsCells
    2. Endocytosis of synaptotagmin 1 is mediated by a novel, tryptophan-containing motif. Traffic. 2003 Jul; 4(7):468-78. Jarousse N, Wilson JD, Arac D, Rizo J, Kelly RB. PMID: 12795692.
      View in: PubMed   Mentions: 14     Fields:    Translation:HumansAnimalsCells
    3. Internalization signals in synaptotagmin VII utilizing two independent pathways are masked by intramolecular inhibitions. J Cell Sci. 2003 Apr 01; 116(Pt 7):1327-37. Dasgupta S, Kelly RB. PMID: 12615974.
      View in: PubMed   Mentions: 5     Fields:    Translation:AnimalsCells
    4. Intersectin 1L guanine nucleotide exchange activity is regulated by adjacent src homology 3 domains that are also involved in endocytosis. Mol Biol Cell. 2003 Apr; 14(4):1624-37. Zamanian JL, Kelly RB. PMID: 12686614; PMCID: PMC153127.
      View in: PubMed   Mentions: 26     Fields:    Translation:AnimalsCells
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